By eliminating HK2, quizartinib and the autophagy inhibitor may prevent cancer cells from metabolizing absorbed glucose and mobilizing stored nutrients, thereby triggering cancer cell death.
CDK inhibitors can block the impact of cyclin E/CDK action, helping restore the beneficial effects of the TGF-beta/SMAD3 pathway, and thus facilitating cancer cell death.
但是当将研究性CFH抗体加入到利妥昔单抗中时,11名患者中有5名(45%)癌细胞死亡明显增加。
When the researchers added CFH antibody to rituximab, five of the 11 patients(45 percent) demonstrated a significant increase in the death of cancer cells.
但是当将研究性CFH抗体加入到利妥昔单抗中时,11名患者中有5名(45%)癌细胞死亡明显增加。
But when the investigational CFH antibody was added to rituximab, five of the 11 patients(45 percent) had a significant increase in cancer cell death.
As more and more cancer cells die, the tumour is stopped from growing larger.
他们也可能促进新的癌细胞死亡,抑制其蔓延。
They may also promote the death of new cancer cells and inhibit their spread.
在两个小时内,Gdovin估计高达95%的癌细胞死亡。
With this method, Gdovin estimated, up to 95 per cent of the targeted cancer cells die within two hours.
在两个小时内,Gdovin估计高达95%的癌细胞死亡。
Within two hours, Gdovin estimates up to 95 per cent of the targeted cancer cells are dead.
这种治疗将导致立即和免疫原性的癌细胞死亡,”他说。
The treatment will cause immediate and immunogenic cancer cell death,” he said.
之后我们发现,24小时之内,竟然有80%到90%的癌细胞死亡。
And what we found was that in 24 hours, 80% to 90% of these cancer cells actually died.”.
然而,更保守和有序的包装密度与化疗引起的更大的癌细胞死亡有关。
A more conservative and ordered packing density, however, was linked to greater cancer cell death in response to chemotherapy.
在DNA被损坏到足以使癌细胞死亡之前需要数天或数周的治疗。
It takes days or weeks of treatment before DNA is damaged enough for cancer cells to die.
这需要发现与MCL1高度紧密和有选择性结合的分子,以充分诱导基于机制的靶向癌细胞死亡。
This necessitates the discovery of a molecule that binds extremely tightly and selectively to MCL1 in order to sufficiently induce on-target, mechanism-based cancer cell death.
可可抗氧化剂-包括表儿茶素和儿茶素-有助于减少炎症,防止癌细胞扩散,并诱导某些癌细胞死亡。
Cocoa antioxidants- including epicatechins and catechins- help reduce inflammation, prevent the spread of cancer cells, and induce death in certain cancer cells.
他说,“所以多年来,研究人员都希望能够弄清BCL-2在过渡活跃时是如何防止癌细胞死亡的。
So for many years researchers were able to figure out what BCL-2 did when it was overactive it prevented cancer cells from dying.”.
ADI-PEG 20 is designed to deplete the external supply of arginine, causing arginine-dependent cancer cells to die while leaving the patient's normal cells unharmed.
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