Global Phase 3 trial of tislelizumab in patients with previously untreated advanced hepatocellular carcinoma(HCC); and.
在接下来的30天里,这些健康细胞开始增殖,取代病变肝细胞,最终占据约三分之一的肝细胞。
Over the next 30 days, those healthy cells began to proliferate and replace diseased liver cells, eventually accounting for about one-third of all hepatocytes.
这使得他们能够评估超过3,800种化合物对与2型糖尿病相关的肝细胞中的基因表达变化的影响。
This allowed them to assess the effects of more than 3,800 compounds on gene expression changes in liver cells that are associated with type 2 diabetes.
在一系列实验中,他们发现缺乏TAK1的幼小动物的肝细胞死亡率很高。
In a series of experiments, they found a high rate of liver cell death in young animals lacking TAK1.
Maryland company Orgenesis(ORGS) is developing a proprietary therapeutic platform that transforms adult liver cells into insulin-generating cells to provide patients with independent insulin production.
In the mice with cancerous tumors almost all hepatocytes showed STAT3 activation, whereas less than two percent of mice without tumors displayed activated(phosphorylated) STAT3.
对肝癌最常见的肝细胞癌(HCC)的综合分析揭示了可能代表新的治疗靶点的几种基因组变化。
An extensive analysis of hepatocellular carcinoma(HCC), the most typical kind of liver cancer, has revealed several genomic changes that could represent new therapeutic targets.
They found that nearly all hepatocytes showed STAT3 activation in mice with cancer, compared to less than two percent of hepatocytes in mice without tumors.
对肝癌最常见的肝细胞癌(HCC)的综合分析揭示了可能代表新的治疗靶点的几种基因组变化。
A comprehensive analysis of hepatocellular carcinoma(HCC), the most common type of liver cancer, has revealed several genomic changes that may represent new therapeutic targets.
They found that nearly all hepatocytes showed STAT3 activation in mice with cancer, compared to less than two percent of hepatocytes in mice without tumors.
起初,肝脏是能够取代垂死的肝细胞。
At first, the liver is able to replace the dying liver cells.
某些类型的肝细胞功能失调AAT积累,这是哪里生产的。
Certain types of dysfunctional AAT accumulate in the liver cells, where it is produced.
因此很有必要寻找替代来源的人的肝细胞,绕过这些问题。
Therefore it has been necessary to find alternative sources of human hepatocytes which circumvent these issues.
渐渐地,分化的肝细胞的再生能力被耗尽,并且形成更多的结缔组织。
Gradually, the regeneration capacity of the differentiated liver cells is exhausted and more connective tissue is formed.
多余的脂肪会在你的肝细胞中堆积,导致非酒精性脂肪性肝病(NAFLD)。
The extra fat can build up in your liver cells and lead to non-alcoholic fatty liver disease(NAFLD).
在当前的研究中,科学家们想要寻找由节食引起的肝细胞基因活动的差异。
In the current study, the scientists looked for liver cell genetic activity differences that were caused by fasting.
Hepatx的创始人正在开发一种再生解决方案,能够培育用于治疗目的的肝细胞。
The founders of Hepatx are developing a regenerative solution enabling hepatocyte production for therapeutic purposes.
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