Examples of using Synaptic cleft in English and their translations into Portuguese
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The release of acetylcholine in the synaptic cleft through the medial olivocochlear efferent tract modulates the motion of outer hair cells.
And the synaptic cleft is the sky between…- the storm
The terminals release transmitter substances into a gap called the synaptic cleft between the terminals and the dendrites of the next neuron.
Once released on the synaptic cleft acetylcholine molecules occupy muscular acetylcholine receptors besides other neuronal receptors, and in special situations the extrajunctional receptors.
an enzyme that degrades the neurotransmitter acetylcholine after it is released into the synaptic cleft.
Tricyclic antidepressants amytriptiline potentiate the actions of noradrenaline by inhibiting its reuptake in the synaptic cleft, which increases the activity of the neuronal circuit.
accumulating in the synaptic cleft during wakefulness and acting as a local inhibitor.
And then the main side effect-- the reason why all this is happening-- is it allows those neurotransmitters to be dumped into the synaptic cleft.
increases the concentration of acetylcholine in the synaptic cleft, antagonized this blockade.
subsequent neurotransmitter release from the motor neuron into the synaptic cleft.
since it is the major control mechanism of serotoninergic function by the reuptake of serotonin in the synaptic cleft.
the release of acetylcholine in the synaptic cleft is normal,
It is possible that this initial increase in the concentration of the neurotransmitter in the synaptic cleft and its prolonged contact with the end-plate terminal could cause desensitization of those receptors
resulting in damage through the toxicity caused mainly by an accumulation of glutamate in the synaptic cleft.
which decreases the exocytosis of noradrenaline in the synaptic cleft, both in the central and peripheral nervous system, it is now prescribed for hypertension management.
A build-up of acetylcholine in the synaptic cleft, due to the inhibition of cholinesterase,
The medial olivocochlear tract modulates the movement of the outer hair cells by the release of acetylcholine in the synaptic cleft, causing a hyperpolarization,
as CNS metabolism decreases along with axonal conduction velocity and changes to the synaptic cleft are observed.
Ach is released into the synaptic cleft and plays its role in cardiac muscle when an important event of the cholinergic neurotransmission must happen:
the primary cell type responsible for the removal of glutamate from the synaptic cleft, in the process of toxicity in animal model.