Voorbeelden van het gebruik van Anandamide in het Engels en hun vertalingen in het Nederlands
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Anandamide is one these endogenous compound manufactured in the brain.
The naturally occurring enzyme FAAH acts to deactivate anandamide.
Our brains and the rest of our body function better at higher anandamide levels.
CBD in turn is capable of influencing anandamide levels in our body.
A well known example is anandamide.
Researchers used the anandamide transporter inhibitor VDM11.
In these people, anandamide breaks down more slowly, so they are generally more relaxed.
The endocannabinoids 2-AG and anandamide as well as a synthetic cannabinoid(WIN55212-2)
Inhibitors of anandamide degradation or re-uptake were shown to reduce colon inflammation in mice and rats.
Science/Cells: Endocannabinoids show activity against resistant bacteria The endocannabinoids anandamide and arachidonoyl serine reduced metabolic activity of the biofilm formation by MRSA methicillin-resistant Staphylococcus aureus.
elevated anandamide and palmitoylethanolamide levels in cerebrospinal fluid,
The endocannabinoids anandamide and palmitylethanolamide inhibited the RTX-induced release of two neuropeptides, that play an important role in the development of neuropathic hyperalgesia.
Science: Immune system Italian researchers demonstrated that anandamide suppresses the release of cytokines such as interleukin-2
If degradation was inhibited and additional anandamide was injected to the animals vomiting was suppressed completely.
Authors suppose that anandamide activates the receptors“CB2,
Acute activation of cannabinoid receptors by anandamide reduces gastro-intestinal motility
In the whole human body, receptors are present for Anandamide, especially in the brain.
Science: Vanilloid receptors The endogenous cannabinoid anandamide but not 2-arachidonyl glycerol activated vanilloid receptors in the hippocampus.
It will not only increase the concentration of anandamide, but also the concentrations of other fatty acid amides.
Moreover, anandamide dose-dependently inhibited the growth of colon cancer cells, which was blocked by a CB1 receptor antagonist.