Examples of using Gnrh in English and their translations into Hungarian
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Thus a single hormone, GnRH, controls a complex process of follicular growth,
GnRH normally stimulates the pituitary gland at the base of the brain to produce two hormones which cause the testes to produce testosterone.
The pulsatility of GnRH secretion has been seen in all vertebrates, and it is necessary to ensure a correct reproductive function.
GnRH agonists work by initially over stimulating the pituitary then rapidly desensitizing it to the effects of GnRH.
Structural lesions of hypothalamus, such as tumors can interfere in the normal pattern of GnRH secretion.
wash the exposed area immediately, as GnRH analogues may be absorbed through the skin.
But, she adds, she wouldn't automatically rule out ART with a GnRH agonist if it were your only choice.
influence the production of the GnRH hormone, which, in turn,
has no castration side effects compared to GnRH agonists.
Clomiphene citrate is a substance that can effectively increase the secretion of gonadotrophin releasing hormone GnRH, which in turn has a positive effect on the production of LH
triptorelin(these medicines are GnRH agonists: they act on the secretion of LH by stimulating the production of GnRH to such an extent that the body stops making LH).
Shuniu GnRH(suitable for dairy cattle)).
show the neurokinin pathway plays an important role both in‘mini-puberty' as well as the GnRH activation in puberty.
In dogs, administration of higher doses of GnRH agonists over a period of time desensitises the pituitary gland with the result that release of gonadotrophins is suppressed, leading to a suppression of reproductive function.
patients develop this unique combination of GnRH deficiency and anosmia(due to loss of olfactory neurons),
Unlike GnRH agonists, GnRH antagonists do not induce a LH surge with subsequent testosterone surge/ tumour stimulation and potential symptomatic flare
1 ng/ ml) was 1.2% compared to 0.8% during GnRH agonist treatment.
was 12% in Orgalutran treated patients and 25% in patients treated subcutaneously with a GnRH agonist.
by studying a single FGFR1 mutation, FGF8 was then identified as the ligand responsible for GnRH neuronal migration
The chemical castration of these children prior to puberty with GnRH agonists(puberty blockers which cause infertility,