Examples of using Synthase in English and their translations into Romanian
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Medicine
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Colloquial
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Official
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Ecclesiastic
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Ecclesiastic
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Computer
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Programming
ATP synthase is involved in the mitochondrial generation of ATP,
However, polyglutamated methotrexate inhibits thymidylate synthase and dihydrofolate reductase,
Hyperammonaemia may be due to the lack of a specific liver enzyme N- acetylglutamate synthase.
Anidulafungin selectively inhibits 1,3-β-D glucan synthase, an enzyme present in fungal,
the protons will spin the ATP synthase which in turn will create ATP.
blood levels of ammonia) in patients who lack an enzyme called N-acetylglutamate synthase(NAGS).
The safety and efficacy of RAVICTI for the treatment of patients with N-acetylglutamate synthase(NAGS) and CITRIN(citrullinaemia type 2)
increases nitric oxide synthase activity in the cerebral cortex.
Metformin stimulates intracellular glycogen synthesis by acting on glycogen synthase and increases the transport capacity of specific types of membrane glucose transporters(GLUT-1 and GLUT-4).
Xanthohumol is a prenylated chalconoid, biosynthesized by a type III polyketide synthase(PKS) and subsequent modifying enzymes.
Bedaquiline specifically inhibits mycobacterial ATP(adenosine 5'-triphosphate) synthase, an essential enzyme for the generation of energy in Mycobacterium tuberculosis.
Thai TV, and religion should be viewed via WiFi 3G synthase year before.
Finally, nitric oxide is another endogenous mediator produced by the mastcell by means of an enzymatic system called NOS, nitric oxide synthase.
works by specifically blocking an enzyme inside the M. tuberculosis bacteria called ATP synthase, which the bacteria need to generate energy.
Nitisinone treatment leads to normalised porphyrin metabolism with normal erythrocyte PBG- synthase activity and urine 5-ALA,
In vitro studies have shown that pemetrexed behaves as a multitargeted antifolate by inhibiting thymidylate synthase(TS), dihydrofolate reductase(DHFR),
which causes a sustained inhibition of ATP synthase, were more resilient to neuronal death after excitotoxic damage.
Experimental studies suggest that reduced endothelial NO levels are mechanistically linked to the uncoupling of endothelial nitric oxide synthase(eNOS) from its essential co-factor tetrahydrobiopterin(BH4),
Acquired resistance mechanisms that affect bedaquiline MICs include mutations in the atpE gene, which codes for the ATP synthase target, and in the Rv0678 gene, which regulates the expression of the MmpS5-MmpL5 efflux pump.
in which an increase in IF1 in the mice reduced the activity of ATP synthase(specifically ATP5A) and was neuroprotective.