Examples of using Kinase in English and their translations into Vietnamese
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Toxicology at the Vetmeduni Vienna, the first author of the study, the myeloid leukaemia cells growth depends on an activating mutation in the FLT3 tyrosine kinase and the cancer cells die if FLT3 is blocked.
activates myosin light-chain kinase, that will enable the myosin crossbridge to bind to the actin filament
prevents diet-induced obesity by activating the enzyme called AMP-activated protein kinase(AMPK).
Sunitinib also inhibits CD117(c-KIT),[1] the receptor tyrosine kinase that(when improperly activated by mutation) drives the majority of gastrointestinal stromal cell tumors.[3] It has been
sensor structure that emerges from the cell, or to target a kinase to a specific part of the cell.
Imatinib targets different tyrosine kinases, depending on the type of cancer.
One of the functions of these Tec tyrosine kinases is to enhance Ca2+ signalling by maintaining the activity of phospholipase Cγ(PLCγ).
Crizotinib has an aminopyridine structure, and functions as a protein kinase inhibitor by competitive binding within the ATP-binding pocket of target kinases.
Imatinib(brand names Gleevec and Glivec) is a drug able to bind the catalytic cleft of these tyrosine kinases, inhibiting its activity.
They can also increase current flow density through the channel.[1] There are also many protein kinases that regulate ASIC function through phosphorylation.
Protein kinases can become mutated, stuck in the“on” position,
Furthermore, tyrosine kinases function in many signal transduction cascades wherein extracellular signals are transmitted through the cell membrane to the cytoplasm
and ERK1/2 kinases.
the Abl protein tyrosine kinases as a consequence of fusion to diverse partner proteins or constitutive production of PDGF have been
AMP-activated protein kinases, cAMP, and Ca(2+)-dependent channels.
The distinguishing feature between janus kinase 2 and other JAK kinases is the lack of Src homology binding domains(SH2/SH3) and the presence of up to seven JAK homology domains(JH1-JH7).
AMP- activated protein kinases, cAMP, and Ca(2+)- dependent channels.
This process involves a leaf-vascular tissue located LRR receptor kinases(LjHAR1, GmNARK and MtSUNN), CLE peptide signalling,
extracellular levels, which, in turn, allows for an accelerated stimulation of glucose uptake and increase protein kinases in skeletal muscle tissue.
on 15 August 2019.[3] It is a selective tyrosine kinase inhibitor(TKI), of the tropomyosin receptor kinases(Trk) A, B and C, C-ros oncogene 1(ROS1) and anaplastic lymphoma kinase(ALK).[4].