Examples of using Mitochondrial dysfunction in English and their translations into Portuguese
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Recent studies have shown that mitochondrial dysfunction is a key factor in development
Some of the many potential mechanisms involved in this impairment are oxidative stress, mitochondrial dysfunction, endoplasmic reticulum stress,
Endoplasmic reticulum stress and mitochondrial dysfunction, caused, in part, by increased metabolic demand,
Besides, mitochondrial dysfunction results in ATP depletion
Functional studies show that the C1494T mutation causes a mild mitochondrial dysfunction and sensitivity to aminoglycosides,
Mitochondrial dysfunction associated with sepsis can reduce the effective utilization of oxygen by cells despite adequate perfusion,
autonomic neuropathy and mitochondrial dysfunction, common in DM, are adjuvant factors in reducing energy formation for adequate heart performance.
functional cell changes, such as mitochondrial dysfunction.
should be fully investigated for possible mitochondrial dysfunction in cases which have relevant signs or symptoms.
Moreover, high levels of ScvO2 indicate increased oxygen delivery- exceeding tissue requirements- a decrease in cellular oxygen consumption mitochondrial dysfunction, and/or a large arteriovenous shunt.
modulates the concentration of free mitochondrial coenzyme a, mitochondrial dysfunction attenuating present in ischemic heart failure.
reactive oxygen species ROS production, mitochondrial dysfunction, and neutrophilic infiltration.
source of ros in the cells, the present study tested the hypothesis that mitochondrial dysfunction leads to the loss of the anticontractile effects of pvat in obesity.
rats lung cells have been suggested that mitochondrial dysfunction and oxidative stress play important role in the amiodarone cytotoxicity.
Therefore, the objective of this study was to evaluate the ability of phenolic compounds catechin and epicatechin to minimize the mitochondrial dysfunction and oxidative damage induced by amiodarone in human lung fibroblast cells mrc-5.
A recent in vitro study with dorsal root ganglion cells of rats has shown that lidocaine-triggered neurotoxicity is related to mitochondrial dysfunction with activation of apoptosis pathways.
the rapid achievement of energy compared to oxidative phosphorylation; mitochondrial dysfunction; or even the maintenance of mitochondrial homeostasis,
animal experimental approaches to examine defective intracellular signaling pathways underlying mitochondrial dysfunction, and related mechanisms of neurodegeneration,
such as dysfunction of the pyruvate dehydrogenase enzyme, mitochondrial dysfunction, and the presence of hepatic or renal dysfunction. .
decreased skeletal muscle adenosine triphosphate content and mitochondrial dysfunction.