Examples of using Cell surface in English and their translations into Slovenian
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Medicine
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Colloquial
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Official
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Ecclesiastic
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Financial
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Computer
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Official/political
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Programming
thereby increasing the quantity of functional CFTR at the cell surface.
Finally, the binding of the"plastic antibodies" to EGFR led to a decrease in the density of receptors on the cell surface.
EGF acts by binding to EGF receptor(EGFR) on the cell surface and stimulating the intrinsic protein-tyrosine kinase activity of the receptor.
then certain proteins displayed on the cell surface.
Interferons exert their cellular activities by binding to specific membrane receptors on the cell surface.
The small amount of F508del-CFTR that reaches the cell surface has low channel-open probability(defective channel gating).
The immunoregulatory activities of granulocyte colony stimulating factor concerns notably cells of the neutrophilic granulocyte lineage which bear cell surface receptors for the protein.
Sebelipase alfa binds to cell surface receptors via glycans expressed on the protein
Once the antibody binds to the cell surface, they can activate specific proteins,
These proteins with short oligosaccharide chains are involved in cell surface recognition by hormones,
After EPO binds to its cell surface receptor, it activates signal transduction pathways that interfere with apoptosis
blocks its interaction with the p55 and p75 cell surface TNF receptors.
neutralizes the biological function of TNF by blocking its interaction with the p55 and p75 cell surface TNF receptors.
IL-23 that is pre-bound to IL-12Rβ 1 cell surface receptors.
IL-23 that is already bound to IL-12R1 cell surface receptors.
by decreasing drug permeability and/or increasing active efflux(pumping out) of the drugs across the cell surface.
blocks its interaction with p55 and p75 cell surface TNF receptors.
blocks its interaction with the p55 and p75 cell surface TNF receptors.
meaning that it blocks particular channels on the cell surface, called calcium channels,
The mechanism of action of etanercept is thought to be its competitive inhibition of TNF binding to cell surface TNFR, preventing TNF-mediated cellular responses by rendering TNF biologically inactive.